S 7 C-D showed that an obvious trend was observable in Luminal &RT, Non-luminal &RT, and Non-luminal &NRT subgroups, but not in Luminal &NRT.
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ALG3 contributes to stemness and radioresistance through regulating glycosylation of TGF-β receptor II in breast cancer.
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S 7 H showed a trend for the subgroup TP53(−) & ALG3(Low) but it was not significant for outcome after RT in the other subgroups, which was consistent with that both TP53 mutation [ 85 ] and ALG3 overexpression would significantly influence the radiosensitivity of breast cancer.