Deletion of <i>Rptor</i> in Preosteoblasts Reveals a Role for the Mammalian Target of Rapamycin Complex 1 (mTORC1) Complex in Dietary-Induced Changes to Bone Mass and Glucose Homeostasis in Female Mice.

In contrast, for Rptor ob −/− mice, HFD feeding resulted in significantly higher trabecular and cortical bone thickness (although trabecular number showed a trend toward reduction) in the proximal tibia compared to the CD‐fed mice.

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