4 A, MTT assays after sunitinib treatment demonstrated that tumor cell growth was inhibited by sunitinib in all RCC cells (RCC-24, 786-O, and Caki-2) and normal HEK 293-T cells in a dose-dependent manner while KTZ did not show a significant trend.
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Repurposing ketoconazole as an exosome directed adjunct to sunitinib in treating renal cell carcinoma.
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