Most of the previous reports using the same mouse model found no difference in oxygenation at baseline, while other studies showed a trend toward lower SO2 and pO2 in sickle mice compared to sickle trait mice rather than normal hemoglobin mice, but the difference was not statistically significant [ 34 , 54 ].
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Chlorine inhalation induces acute chest syndrome in humanized sickle cell mouse model and ameliorated by postexposure hemopexin.
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