Postnatal prolongation of mammalian nephrogenesis by excess fetal GDNF.

Development · 2021 · PMC8180252 · PMID 34032268

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Further analysis of the WNT/β-catenin signaling targets ( Clevers and Nusse, 2012 ) in E14.5 control and Gdnf hyper/hyper kidneys demonstrated significant upregulation of canonical target Axin2 ( P =0.003371), whereas NP-expressed WNT targets ( Karner et al., 2011 ) showed a trend of downregulation, likely reflecting the reduced overall number of NPCs ( Fig.

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