Programmed suppression of oxidative phosphorylation and mitochondrial function by gestational alcohol exposure correlate with widespread increases in H3K9me2 that do not suppress transcription.

The Satb1 promoter displayed increased H3K9me2, and although the Satb2 promoter trended toward an increase, this did not reach statistical significance ( p = 0.1) and was only different from samples derived from EtOH-exposed unaffected mice.

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