Genome instability and pressure on non-homologous end joining drives chemotherapy resistance via a DNA repair crisis switch in triple negative breast cancer.

There was significant loss in chromosome count in the MDA-MB-231 (Figure 1F ) and BT549 (Figure 1G ) and trended lower in Hs578t (Figure 1H ) and MDA-MB-468 cells (Figure 1I ) but did not reach statistical significance.

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