IFN-β Acts on Monocytes to Ameliorate CNS Autoimmunity by Inhibiting Proinflammatory Cross-Talk Between Monocytes and Th Cells.

Splenocytes from rIFN-β-treated mice activated with myelin Ag secreted less GM-CSF and IL-17A in culture supernatants, whereas reduction in IFN-γ secretion did not reach statistical significance ( Supplementary Figure 1B ).

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