Further analysis showed a trend towards an increase in complement component C5a independent of component C3, observed to be tightly regulated by complement factor H.
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Oxidative Stress and Mitochondrial Damage in Dry Age-Related Macular Degeneration Like <i>NFE2L2/PGC-1α</i> <i><sup>-/-</sup></i> Mouse Model Evoke Complement Component C5a Independent of C3.
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