3a–e ), while S100A10 , SLC10A6 , B3GNT5 , CD14 , CD109 , and EMP1 each showed various degrees of upregulation that did not reach statistical significance (Supplementary Fig. 3f–k ).
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Fibrillar α-synuclein induces neurotoxic astrocyte activation via RIP kinase signaling and NF-κB.
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