Dipeptidyl peptidase 4 contributes to Alzheimer's disease-like defects in a mouse model and is increased in sporadic Alzheimer's disease brains.

Nevertheless, although it did not reach statistical significance, we observed a consistent reduction of Aβ42-positives plaques ( Fig. 5 , A and B ), mean area ( Fig. 5 C ) and perimeter ( Fig. 5 D ).

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