7.0 months 95%CI: 0.0–15.2 in KRAS -wild-type; p = 0.100), this trend in OS2L did not reach statistical significance ( p = 0.100; Figure S8 ).
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Silibinin Suppresses Tumor Cell-Intrinsic Resistance to Nintedanib and Enhances Its Clinical Activity in Lung Cancer.
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Accordingly, highly significant, supra-additive increases in cell doubling times were observed in those cells simultaneously exposed to nintedanib and silibinin ( Figure S7 , bottom panels ). 2.8.