Comparison of SHANK3 deficiency in animal models: phenotypes, treatment strategies, and translational implications.

J Neurodev Disord · 2021 · PMC8594088 · PMID 34784886

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Although most changes of protein levels showed a trend towards reduction, increased levels of the cell adhesion proteins CNTN2 and CDH4, MYEF2, which serves as a transcriptional repressor of the myelin basic protein, the Akt-regulating E3 ubiquitin-protein ligase TTC3, and members of the ribosomal protein family have been observed in striatal synapses [ 202 ].

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