Antibody profiling of bumble mice that received PerC transfer showed a trend of increased anti- T . gondii antibody generation following chronic infection ( Fig 4B ), but antibody reactivity to the parasite did not reach levels observed in wildtype mice, suggesting the B-1 compartment has a limited role in generating high-affinity parasite-specific antibodies.
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Genetic mapping reveals Nfkbid as a central regulator of humoral immunity to Toxoplasma gondii.
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