HuR drives lung fibroblast differentiation but not metabolic reprogramming in response to TGF-β and hypoxia.

Although there was an elevated tendency in HKII expression under conditions of hypoxia and TGF-β1, this did not reach statistical significance (Fig. 2 b).

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We did not find any significant changes with hypoxia alone, although we noted a slight trend in increased lactate compared with normoxia.

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