However, when we evaluated density of Iba‐1+ microglia specifically in infarct‐adjacent areas (termed ROI, see Section 2 ), we recorded a significant effect of exendin‐4 in reducing Iba‐1+ cell density in these regions for non‐T2D compared with T2D‐Ex‐4, with a strong trend for T2D‐Ve compared with T2D‐Ex‐4 (Figure 4b ).
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Normalisation of glucose metabolism by exendin-4 in the chronic phase after stroke promotes functional recovery in male diabetic mice.
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