PK pre-treatment showed a trend in reducing AβO and TauO binding to human FC and HP synaptosomes, especially at the 2.5 μM and 5 μM concentrations of both oligomers (Supplementary Fig. 6, Additional file 1).
← all excerpts
Aβ/tau oligomer interplay at human synapses supports shifting therapeutic targets for Alzheimer's disease.
1
—
—