Several proteins involved in LTP were reduced following SAH, including Ca 2+ /calmodulin- dependent protein kinase II (CamK II), myelin basic protein (MBP), along with a marked trend towards reduced GluR1 and increased glutamate receptor type 2 (GluR2) [ 437 ].
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The blood-brain barrier and the neurovascular unit in subarachnoid hemorrhage: molecular events and potential treatments.
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