Remarkably, the most affected metabolic substrates were those related to glycolysis − glucose-6-phosphate (G6P) and lactate levels showed a trend toward a decrease in the EAAm-treated cells ( Figure 4 B), thus suggesting downregulation of glycolytic flux.
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An amino acid-defined diet impairs tumour growth in mice by promoting endoplasmic reticulum stress and mTOR inhibition.
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