Haploinsufficiency of Col5a1 causes intrinsic lung and respiratory changes in a mouse model of classical Ehlers-Danlos syndrome.

In the present study, tissue elasticity (H) trended lower in Col5a1 +/− mice, as anticipated, but did not reach statistical significance.

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Their potential impact on the respiratory function during lung infections, other respiratory disease processes, or insults may be significant and justify further clinical evaluation.

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