Endothelial cells of pulmonary origin display unique sensitivity to the bacterial endotoxin lipopolysaccharide.

LPS treatment demonstrated a decreasing trend in pro‐CDH13 and CDH13 expression in pulmonary HMVECs, but unfortunately, the results were inconclusive and could not be determined with flow cytometry analysis, as the antibody was not suitable for this.

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Upon LPS stimulation, there was a trend towards a decrease in pro‐CDH13 and CDH13 expression in pulmonary ECs following quantification of the Western blots (Figure 4e,f ), however, this did not reach statistical significance and was not immediately visible upon blotting (Figure 4d ). 3.5.

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