MCTR3 reprograms arthritic monocytes to upregulate Arginase-1 and exert pro-resolving and tissue-protective functions in experimental arthritis.

Mass spectrometric analysis demonstrated an upregulation of putrescine in joints from mice receiving MCTR3-reprogrammed monocytes when compared with those that received monocytes incubated with vehicle alone, although this increase did not reach statistical significance ( Figure 6 h).

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