In line with these findings, 4×SUMO2 showed a trend of competing with XRCC4-XLF interaction when added to whole cell extracts at 1 μM concentration (=25 μg) prior to GFP-XRCC4 pulldowns (Figure 6F ).
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Microarray screening reveals two non-conventional SUMO-binding modules linked to DNA repair by non-homologous end-joining.
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