Ketone body and FGF21 coordinately regulate fasting-induced oxidative stress response in the heart.

In addition, Nox4, Ucp2 and Ucp3 tended to be more induced after fasting in Fgf21 −/− mice than WT mice although a difference did not reach statistical significance.

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Nox4 and antioxidant genes such as catalase and Ucp2 showed a highly significant correlation with βOHB concentrations in both WT and Fgf21 −/− mice hearts (Fig. 6 ).

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