Identification of functional pathways and molecular signatures in neuroendocrine neoplasms by multi-omics analysis.

This was relieved, among others, for chromosome 19, where patients harboring amplifications showed a trend to an overall increased gene counts within the same chromosome with respect to not amplified samples (Fig. 3 C), and for chromosome 22, in which an opposite scenario was observed for patients showing deletions (Fig. 3 D).

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