Radiation-response in primary fibroblasts of long-term survivors of childhood cancer with and without second primary neoplasms: the KiKme study.

Only one gene showed a differential reaction after exposure to LDIR: Comparing N2+ and N0, Calmodulin binding transcription activator 2 ( CAMTA2 ) was upregulated with a borderline significant p -value [LFC: 0.101, p -value = 0.056 (crude model)] after LDIR (Table 2 ).

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4 , Additional file 5 c), senescence ( senescence of cells and senescence of fibroblast cell lines ) showed a trend toward activation, interphase [(N0: interphase and interphase of fibroblasts (both, crude model and model 1), N2+ : interphase (all models) and interphase of fibroblasts (crude model)] toward inactivation after HDIR.

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