Targeting <i>PTEN</i> but not <i>SOCS3</i> resists an age-dependent decline in promoting axon sprouting.

Compared with middle-aged WT controls, SOCS3 deleted mice exhibited a trend for more CST sprouting at 50 and 150 μm from the midline, which did not reach statistical significance ( Figure 1 J).

also in 111,027 other papers