Isoforms of Human MARCH1 Differ in Ability to Restrict Influenza A Viruses Due to Differences in Their N Terminal Cytoplasmic Domain.

When examining the RNA copy number ( Figure 5 C), MARCH1.1 showed a trend for increased expression 6 h, but not 24 h, after exposure to IAV or IFN-a and this was significant for IFN-a treatment at 6 h, but not 24 h, when comparing the fold-change relative to mock ( Figure 5 D).

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