Densitometry showed that the difference in signal intensities in this molecular weight range were highly significant (Fig. 5 B).
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Somatostatin slows Aβ plaque deposition in aged APP<sup>NL-F/NL-F</sup> mice by blocking Aβ aggregation.
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The Sst -associated difference in plaque densities was indeed most pronounced at this relatively early timepoint in the size range that included the smallest detectable plaques (0.25–100 μm) but did not reach statistical significance (Fig. 3 G).