Asymmetric dysregulation of glutamate dynamics across the synaptic cleft in a mouse model of Alzheimer's disease.

While this postsynaptic dataset did not reach statistical significance, it is worth pointing out that at 100 pulses, ceftriaxone tended to accelerate glutamate clearance in WT tissue while tending to slow clearance in 3xTg tissue, though the possible explanation for such tendencies are unclear at present.

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