pylori OMV‐treated App NL‐G‐F mice compared to controls as shown by qPCR and immunostaining (Figure S10a‐e ), while only the protein expression level of C3aR showed a trend towards upregulation after H. pylori OMV treatment in App NL‐G‐F mice (Figure S10f‐j ).
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Helicobacter pylori-derived outer membrane vesicles contribute to Alzheimer's disease pathogenesis via C3-C3aR signalling.
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