Receptor-interacting protein kinase 2 (RIPK2) profoundly contributes to post-stroke neuroinflammation and behavioral deficits with microglia as unique perpetrators.

There was also a strong trend toward decreased Iba-1 positivity in the subcortex of Ripk2 −/− brains compared to controls (Fig. 3 C).

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We found that the μKO mice had enhanced capability to travel further distances in the open field test compared to the WT at 24 h and showed a strong trend toward significance at 48 h after injury (Fig. 9 A–D).

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