Sorting nexin 3 exacerbates doxorubicin-induced cardiomyopathy <i>via</i> regulation of TFRC-dependent ferroptosis.

3 O) than those of Dox-treated N-Tg mice, indicating that SNX3 overexpression was associated with reduced cardiac contractile function in Dox-treated mice, although the difference in some echocardiographic parameters did not reach statistical significance ( Fig.

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Furthermore, our data also demonstrate a significant trend toward an increase in cardiac 4-HNE in the SNX3 overexpression plus Dox treatment group compared with Dox treatment alone.

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