Mathematical model calibrated to <i>in vitro</i> data predicts mechanisms of antiviral action of the influenza defective interfering particle "OP7".

NP distribution showed a trend toward longer segments, because they require the binding of more NP for stabilization ( Figure 4 C).

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The related AIC values also show a clear trend toward the three-particle hypothesis ( Table S2 ), making it preferable for the final OP7/STV co-infection model.

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