Neutrophil extracellular traps promote immunopathogenesis of virus-induced COPD exacerbations.

Sputum H3.1 showed trends towards increases at exacerbation onset and 2 weeks that failed to reach statistical significance (Fig. 3C ).

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There was no evidence of neutrophil elastase inhibition leading to downstream activation of pathways promoting neutrophil recruitment or persistence, as neutrophil counts at day 4 were not increased and showed a trend towards remaining reduced (Supplementary Fig 6 ).

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