Furthermore, PV loop data showed a trend towards an increase in LV end-diastolic pressure (EDP) and systolic pressure (ESP) in CKD swine (Fig. 2 e, f).
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Oxidative stress initiates hemodynamic change in CKD-induced heart disease.
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Similar to our findings in myocardial tissue of the swine, this was associated with a lower expression of NNT and GPX3, while the lower levels of SOD2 failed to reach statistical significance (Fig. 6 d–f).