Redox-dependent purine degradation triggers postnatal loss of cardiac regeneration potential.

4B ), and DCA treatment also showed a trend towards an increase in the dATP level ( Supplementary Fig. 4C ), in accordance with the extended cardiomyocyte proliferative window [ 3 ] ( Supplementary Fig. 2 ).

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The activity of G6PD showed a tendency toward reduction, which did not reach statistical significance likely due to considerable variation across the samples ( Fig. 3 E).

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