Acetyl-CoA synthetase 2 alleviates brain injury following cardiac arrest by promoting autophagy in brain microvascular endothelial cells.

Although ACSS2 overexpression exhibited improvements in average vessel length, total number of end points and junctions within the hippocampal region, these changes failed to reach statistical significance (Fig. 2 M-O).

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Although there was a transient increase in serum potassium levels observed 1 h after the intervention, this elevation did not reach statistical significance (Figure S1 G).

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