Dravet syndrome: From neurodevelopmental to neurodegenerative disease?

Finally, there was also a strong trend ( p = .05) for externalizing behaviors with a potential psychotic disorder.

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In brains of patients with AD, Nav1.1 levels were significantly depleted, Nav1.6 levels showed a trend toward depletion, and Nav1.2 levels were unchanged, suggesting that neuronal loss alone cannot explain low levels of Nav1.1. 39 Studies in amyloid precursor protein (APP) and presenelin1 (PS1) animal models of AD also showed that Nav1.1 levels are reduced, confirming the role of SCN1A gene in A

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