Tumor cells upregulate CCL22 in a STING-dependent manner in response to paracrine factors released by STING-activated myeloid cells and type I interferons.

S1 also shows a downward trend in CCL22 expression, although these decreases did not reach statistical significance in the context of a Kruskal–Wallis multiple comparisons test, which generally has less power to identify differences in means compared with the t test used to analyze data in Fig. 1B .

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The neutralizing effect of MMHAR-2 was most noticeable in cells treated with IFN-α, where the P value is approaching significance at 0.0659, and these results parallel those observed for MMHAR-2 inhibition of IFN-α-mediated Stat1 phosphorylation ( Fig.

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