Anti-TLR2 immunotherapy modulates neuron-to-oligodendrocyte propagation of α-synuclein in mouse and human models.

5j, k ) and showed a trend toward reduced microgliosis (Supplementary Fig. 5j, l ) as well.

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Myd88, another TLR2 downstream effector, was also increased in αSyn Tg mice and reduced with NM-101 treatment, though this reduction did not reach statistical significance (Supplementary Fig. 4h, j ).

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