OSBPL3-driven sterol metabolic reprogramming promotes oncogenic signaling and therapeutic resistance in pancreatic cancer.

The correlation between the IC50 values of oxaliplatin and OSBPL3 expression was highly significant ( P = 3.01e-7, R = 0.368) ( Figure S5 A ).

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Principal component analysis (PCA) also showed a trend toward spatial separation, indicating molecular heterogeneity between the groups; however, due to limited sample size and within-group variability, the 95% confidence intervals partially overlapped, and the separation did not reach statistical significance ( Figure S7 G ).

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