Identification of HSPE1 as a new actionable cancer vulnerability leads to an innovative and effective combination therapy for pancreatic ductal adenocarcinoma.

Although stratification based on HSPE1 expression revealed a trend toward reduced survival in high-expressing patients compared to low-expressing counterparts, the differences did not reach statistical significance.

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While a clear trend toward enhanced cytotoxicity is observed, the magnitude of this additional effect is limited.

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