Histone decrotonylation plays a distinct role in HIV latency.

Consistent with prior findings, RGFP966 demonstrated a trend toward enhancing H3K27cr; however, the observed increase did not reach statistical significance.

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Conversely, levels of the crotonylation erasing enzyme chromodomain Y‐like (CDYL) showed a trend of decreasing within 2 to 4 days postactivation but rebounding by day 7, coinciding with HIV latency reestablishment ( Fig. 1A ).

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