Dynamin 2 Regulates Mitochondrial Mitotic Fission in Pulmonary Hypertension.

In contrast, at 96 hours post-siDNM2, there was reduced basal glycolysis and compensatory glycolysis, and a strong trend toward decreased basal respiration, proton leak, and ATP-linked oxygen consumption rate ( Figure S17K through S17S ).

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Silencing DNM2 showed a marked trend toward apoptosis induction under basal, unstimulated conditions, indicating that depletion of DNM2 alone is sufficient to activate intrinsic cell death pathways in PAH PASMCs (Figure 4 F).

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