Reduced ULK1 links impaired autophagy and mitophagy to Alzheimer's disease pathology.

Due to the variations in the data, some of the aforementioned NOR/NOL/Y-maze data did not reach statistical significance (Extended Data Fig. 3 ).

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Furthermore, the ULK1 inhibitor SBI-0206965 showed a trend toward increasing tau puncta formation, whereas the ULK1 inhibitor XST-14 at 5 μM exacerbated intracellular p-tau aggregation but had no effect on degradation of preformed tau foci (Extended Data Fig. 10a,b ).

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