In contrast, MitoCoat treatment resulted in only modest increases in ATP that did not reach statistical significance, despite a significant improvement in cell viability after injury ( Supplementary Figure S1 ) compared to untreated controls.
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Mitochondria-Associated mRNAs Restore ATP During Oxidative Stress via Cytosolic Translation.
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mRNA treatment showed a trend toward restoring these proteins, with PDH and ATP5A significantly increasing ( Figure 5 G,H).