Although the differences did not reach statistical significance—likely due to the limited number of FL_CI cases—we observed a trend toward higher mutation frequencies in RRAGC, ATM, POU2F2, and SPEN, suggesting potential contributors to extranodal lymphomagenesis.
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Genomic heterogeneity in primary cutaneous follicular center lymphomas reveals different clinicopathological subgroups.
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Group 1 tumors also showed a trend toward higher T‐cell infiltration (Figure 4E ), though not statistically significant.