Suppression of inflammatory immune responses in celiac disease by experimental hookworm infection.

There was also an apparent IFN-γ and IL-17A response to QE65 although these failed to reach statistical significance (p = 0.053 and p = 0.073 respectively).

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As shown in Figure 3A , prior to infection or challenge highly significant levels of IL-2 were produced upon stimulation with QE65 compared to medium alone.

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Levels of the TH1 cytokine IFN-γ and the TH17 cytokine IL-17A tended to increase after gluten challenge in the control group only, but this change did not reach statistical significance.

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Production of the regulatory cytokine IL-10 in response to QE65 was significantly increased after gluten challenge, however a small trend for increased levels after challenge, prior to infection, was also seen ( Figure 3G ).

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