Ceftriaxone preserves glutamate transporters and prevents intermittent hypoxia-induced vulnerability to brain excitotoxic injury.

Graphic comparison of ceftriaxone-treated and untreated slices at each glutamate concentration, revealed no significant differences in the RA or SH-exposed slices, although a clear trend of decreased excitotoxicity was apparent in ceftriaxone-treated SH slices ( Fig. 5 ).

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Immunohistochemical stainings showed that EAAT1 and EAAT2 expression significantly decreased after IH and while there was a trend of decrease after SH, it did not reach statistical significance ( Fig. 6 ).

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